梶川 修平助教

専門分野

骨生物学

研究分野

骨生物学
がん

研究内容

‣ 破骨細胞の遊走性制御メカニズムの解明

‣ 骨芽細胞の分化制御メカニズムの解明

‣ がん細胞浸潤制御因子の探索

 

研究業績

  1.  Jumpei Shirakawa*, Shuhei Kajikawa*, Ralph T Böttcher, Mercedes Costell, Yayoi Izu, Tadayoshi Hayata, Masaki Noda, Yoichi Ezura. *These authors contributed equally to this work. Profilin 1 Negatively Regulates Osteoclast Migration in Postnatal Skeletal Growth, Remodeling, and Homeostasis in Mice. JBMR Plus. 3:e10130 (2019).
  2.  Shuhei Kajikawa, Yuu Taguchi, Tadayoshi Hayata, Yoichi Ezura, Ryo Ueta, Sumimasa Arimura, Jun-ichiro Inoue, Masaki Noda, Yuji Yamanashi. Dok-3 and Dok-1/-2 adaptors play distinctive roles in cell fusion and proliferation during osteoclastogenesis and cooperatively protect mice from osteopenia. Biochem. Biophys. Res. Commun. 498, 967-974 (2018)
  3.  Toshiaki Sakamoto, Kei-ichi Ozaki, Kohsuke Fujio, Shu-hei Kajikawa, Shin-ichi Uesato, Kazushi Watanabe, Susumu Tanimura, Takehiko Koji, Michiraki Kohno. Blockade of the ERK pathway enhances the therapeutic efficacy of the histone deacetylase inhibitor MS-275 in human tumor xenograft models. Biochem. Biophys. Res. Commun. 433, 456-462 (2013)
  4.  Ryuichi Mashima, Sumimasa Arimura, Shuhei Kajikawa, Hideaki Oda, Susumu Nakae, Yuji Yamanashi. Dok adaptors play anti-inflammatory roles in pulmonary homeostasis. Genes Cells. 18, 56-65 (2013)

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